Cerebral Venous Thrombosis: NCCT

 History: A 32 yr female c/o sudden headache followed by loss of consciousness. Post partum statusFindings:Multiple ill-defined hypodense lesions are seen in the brain parenchyma bilaterallyNot confined to single arterial teritoryMost of the lesions show hemorrhagic transformationMild surrounding edema is seenHypderdense superior saggital sinus - thrombosisThe findings are typical of Hemorrhagic venous infarctsDiagnosis:Hemorrhagic venous infarctsExtra EdgeEtiologyGenetic prothrombotic conditions, dehydration, trauma, brain surgery, post partum state, OC pills, mastoiditisHow to differentiate arterial from venous infarcts?Arterial infarcts are usually unilateral, few in no and confined to one arterial territory. Hemorrhagic transformation may occur in subacute stage. Arterial occlusion/ thrombosisVenous infarcts are multiple, bilateral and non territorial. Hemorrhage frequent. Venous occlusion/ thrombosisFamous Radiology Blog by Dr Sumer Sethi www.sumersethi.com

Cerebral Venous Thrombosis: NCCT

 History:

 A 32 yr female c/o sudden headache followed by loss of consciousness. Post partum status

Findings:



  • Multiple ill-defined hypodense lesions are seen in the brain parenchyma bilaterally
  • Not confined to single arterial teritory
  • Most of the lesions show hemorrhagic transformation
  • Mild surrounding edema is seen
  • Hypderdense superior saggital sinus - thrombosis

The findings are typical of Hemorrhagic venous infarcts

Diagnosis:

Hemorrhagic venous infarcts

Extra Edge

Etiology

Genetic prothrombotic conditions, dehydration, trauma, brain surgery, post partum state, OC pills, mastoiditis

How to differentiate arterial from venous infarcts?

  • Arterial infarcts are usually unilateral, few in no and confined to one arterial territory. Hemorrhagic transformation may occur in subacute stage. Arterial occlusion/ thrombosis
  • Venous infarcts are multiple, bilateral and non territorial. Hemorrhage frequent. Venous occlusion/ thrombosis